Evidence for retrograde degeneration of epinephrine neurons in Alzheimer's disease
โ Scribed by Dr. William J. Burke; Hyung D. Chung; Jung S. Huang; Shuan S. Huang; John H. Haring; Randy Strong; Gary L. Marchshall; Tong H. Joh
- Publisher
- John Wiley and Sons
- Year
- 1988
- Tongue
- English
- Weight
- 566 KB
- Volume
- 24
- Category
- Article
- ISSN
- 0364-5134
No coin nor oath required. For personal study only.
๐ SIMILAR VOLUMES
In Alzheimer's disease (AD) synapses degenerate and neurons die in brain regions involved in learning and memory processes. Although the cellular and molecular mechanisms underlying the neurodegenerative process in AD are unclear, increasing evidence suggests roles for amyloid โค-peptide (Aโค) and bio
Mitochondria1 defects may be involved in Alzheimer's disease (AD) by lowering the oxidative phosphorylation efficiency at an earlier age. To investigate the possible contribution of an inherited mitochondrial DNA defect to familial AD (FAD), we examined the Genetic Analysis Workshop (GAW) data for e
## Abstract Autophagosomes are accumulated in Alzheimer's disease (AD), but the regulatory pathway of autophagy in AD remains largely unknown. By using electron microscopy, Western blotting, and immunocytochemistry, here we show that autophagosomes are accumulated in rat neurons by okadaic acid (OA