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A biphenotypic transformation of 8p11 myeloproliferative syndrome with CEP1/FGFR1 fusion gene

✍ Scribed by Katsuya Yamamoto; Hiroki Kawano; Shinichiro Nishikawa; Kimikazu Yakushijin; Atsuo Okamura; Toshimitsu Matsui

Book ID
114794077
Publisher
John Wiley and Sons
Year
2006
Tongue
English
Weight
701 KB
Volume
77
Category
Article
ISSN
0902-4441

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## Abstract 8p11 myeloproliferative syndrome (EMS) is a clinical‐pathologic entity characterized by rearrangements involving the __FGFR1__ gene, which encodes a receptor tyrosine kinase. These rearrangements invariably lead to aberrant fusion proteins in which the kinase activity is constitutively

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## Abstract The 8p11 myeloproliferative syndrome (EMS) is an aggressive hematological malignancy caused by the fusion of diverse partner genes to fibroblast growth factor receptor 1 (__FGFR1__). The partner proteins promote dimerization and ligand‐independent activation of __FGFR1__‐encoded tyrosin