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Vitamin E-deficiency in the guinea pig: Effect of succinate on mitochondrial NAD-linked oxidation

✍ Scribed by F.B. Carabello; J.W.C. Bird

Book ID
118851250
Publisher
Elsevier Science
Year
1969
Tongue
English
Weight
356 KB
Volume
34
Category
Article
ISSN
0006-291X

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In both hereditary hemochromatosis (HHC) and in the various forms of secondary hemochromatosis, there is a pathological expansion of total body iron stores, due mainly to an inappropriate increase in absorption of dietary iron (1-3). The liver is the major recipient of this excess absorbed iron, and