𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Unusual morphology in a case of large granular cell leukemia

✍ Scribed by S. Galimberti; R. Riccioni; A. Azzarà; R. Testi; R. Fazzi; C. Testi; M. Petrini


Publisher
Springer
Year
2001
Tongue
English
Weight
68 KB
Volume
80
Category
Article
ISSN
0939-5555

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Aggressive T-cell large granular lymphoc
✍ Todd J. Alekshun; Jianguo Tao; Lubomir Sokol 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 167 KB 👁 2 views

## Abstract The majority of patients with T‐cell large granular lymphocyte (LGL) leukemia will have an indolent clinical course. Herein, we report a case of an aggressive T‐cell LGL leukemia in a previously healthy 42‐year‐old Caucasian male who presented with acute onset of B‐symptoms, hepatosplen

Etiology of neutrophilic granulocytic hy
✍ Friedman, Henry D. ;Goldberg, Jack ;Kurec, Anthony S. ;Davey, Frederick R. 📂 Article 📅 1994 🏛 John Wiley and Sons 🌐 English ⚖ 429 KB

## Abstract The association between large granular lymphocytic leukemia (LGLL) and neutropenia is poorly understood. We attempted to assess whether neutropenia in LGLL with paraproteinemia has a different etiology than LGLL without paraproteinemia. We found neither direct serum inhibition of granul

Large granular lymphocyte leukemia with
✍ Masuda, Michihiko; Arai, Yukari; Nishina, Hiroyuki; Fuchinoue, Shouhei; Mizoguch 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 191 KB 👁 2 views

Neoplastic disorders sometimes accompany a renal transplant. Herein, we report a large granular lymphocyte (LGL) leukemia patient with pure red cell aplasia (PRCA) after renal transplantation. A 36-year-old female was presented to our department with anemia in February 1996. She had undergone hemodi

Unusual cytogenetics in a case of acute
✍ Morse, Helvise G. ;Hays, Taru ;Odom, Lorrie F. 📂 Article 📅 1979 🏛 John Wiley and Sons 🌐 English ⚖ 268 KB

The cytogenetics in the study of a patient with acute lymphoblastic leukemia are presented. Initially, a large proportion of both unstimulated and phytohemagglutinin (PHA)-stimulated blood mitoses showed an abnormal karyotype with a 7;12 translocation and a trisomy 19. At the time of relapse, a PHA-