✦ LIBER ✦

The MYCN oncogene is a direct target of miR-34a

✍ Scribed by Wei, J S; Song, Y K; Durinck, S; Chen, Q-R; Cheuk, A T C; Tsang, P; Zhang, Q; Thiele, C J; Slack, A; Shohet, J

Book ID: 109970473
Publisher: Nature Publishing Group
Year: 2008
Tongue: English
Weight: 283 KB
Volume: 27
Category: Article
ISSN: 0950-9232
DOI: 10.1038/onc.2008.154

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Glutaredoxin is a direct target of oncog

Glutaredoxin is a direct target of oncogenic jun

✍ Goller, Martin E; Iacovoni, Jason S; Vogt, Peter K; Kruse, Ulrich 📂 Article 📅 1998 🏛 Nature Publishing Group 🌐 English ⚖ 737 KB

The TWIST1 oncogene is a direct target o

The TWIST1 oncogene is a direct target of hypoxia-inducible factor-2α

✍ Gort, E H; van Haaften, G; Verlaan, I; Groot, A J; Plasterk, R H A; Shvarts, A; 📂 Article 📅 2007 🏛 Nature Publishing Group 🌐 English ⚖ 412 KB

The miR-15a–miR-16-1 cluster controls pr

The miR-15a–miR-16-1 cluster controls prostate cancer by targeting multiple oncogenic activities

✍ Bonci, Désirée; Coppola, Valeria; Musumeci, Maria; Addario, Antonio; Giuffrida, 📂 Article 📅 2008 🏛 Nature Publishing Group 🌐 English ⚖ 578 KB

miR-92 is a key oncogenic component of t

miR-92 is a key oncogenic component of the miR-17–92 cluster in colon cancer

✍ Akihiko Tsuchida; Shinichiro Ohno; Weihong Wu; Nariso Borjigin; Koji Fujita; Tas 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 898 KB

p53-independent upregulation of miR-34a

p53-independent upregulation of miR-34a during oncogene-induced senescence represses MYC

✍ Christoffersen, N R; Shalgi, R; Frankel, L B; Leucci, E; Lees, M; Klausen, M; Pi 📂 Article 📅 2009 🏛 Nature Publishing Group 🌐 English ⚖ 445 KB

Circular dichroism study of DNA binding

Circular dichroism study of DNA binding by a potential anticancer peptide nucleic acid targeted against the MYCN oncogene

✍ Andrea Faccini; Andrea Tortori; Tullia Tedeschi; Stefano Sforza; Roberto Tonelli 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 255 KB

## Abstract The interaction with DNA of a peptide nucleic acid (PNA) oligomer (16nt) conjugated with a nuclear localization signal (NLS) peptide, which was previously found to be able to inhibit tumor cell proliferation through block of transcription of the __MYCN__ oncogene, was studied by UV and