✦ LIBER ✦

Recessive oncogenes

✍ Scribed by Robert Bookstein; D. Craig Allred

Publisher: John Wiley and Sons
Year: 1993
Tongue: English
Weight: 862 KB
Volume: 71
Category: Article
ISSN: 0008-543X
DOI: 10.1002/1097-0142(19930201)71:3+<1179::aid-cncr2820711442>3.0.co;2-b

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Transcription factors and recessive onco

Transcription factors and recessive oncogenes in the pathogenesis of human lung cancer

✍ John D. Minna; Jochen Schütte; Jean Viallet; François Thomas; Frederic J. Kaye; 📂 Article 📅 1989 🏛 John Wiley and Sons 🌐 French ⚖ 421 KB 👁 1 views

Expression of the jun family of transcription factorslproto-oncogenes in lung cancer From recent studies it is now clear that the product of the cellular counterpart of the oncogene jun (Maki et al., 1987) is one of the proteins making up the transcription-factor preparation AP-1 (Angel et al., 198

Oncogenes and proto-oncogenes

✍ J. Michael Bishop 📂 Article 📅 1986 🏛 John Wiley and Sons 🌐 English ⚖ 670 KB

Oncogenes

✍ John Paul 📂 Article 📅 1984 🏛 John Wiley and Sons 🌐 English ⚖ 696 KB

Studies in oncogenes

✍ Nigel K. Spurr 📂 Article 📅 1991 🏛 Elsevier Science 🌐 English ⚖ 550 KB

Advances are being made at an increasing rate towards an understanding of the role of cellular oncogenes in the normal and transformed cell. This review highlights a number of studies looking at the role of these genes in the carcinogenic process using in vivo and in vitro systems. Current Opinion

Insertion mutation inactivates the expre

Insertion mutation inactivates the expression of the recessive oncogene lethal(2)giant larvae of Drosophila melanogaster

✍ Lützelschwab, R. ;Müller, G. ;Wälder, B. ;Schmidt, O. ;Fürbass, R. ;Mechler, B. 📂 Article 📅 1986 🏛 Springer 🌐 English ⚖ 701 KB

Basic science reviews: Oncogenes

✍ Dr. Norris K. Lee 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 879 KB

Currently, the most widely accepted paradigm of oncogenesis is based on the concept of a balance between positive-and negative-growth regulatory mechanisms. Aberrations in these regulatory pathways can lead to a transformed phenotype, seen clinically as tumorigenesis. In vivo, there may be additiona