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Protein–protein interactions in the assembly and subcellular trafficking of the BACE (β-site amyloid precursor protein-cleaving enzyme) complex of Alzheimer's disease

✍ Scribed by Parsons, R.B.; Austen, B.M.

Book ID
120484756
Publisher
Portland Press
Year
2007
Tongue
English
Weight
320 KB
Volume
35
Category
Article
ISSN
0300-5127

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## Abstract One of the pathologic hallmarks of Alzheimer's disease is the excessive deposition of β‐amyloid peptides (Aβ) in senile plaques. Aβ is generated when β‐amyloid precursor protein (APP) is cleaved sequentially by β‐secretase, identified as β‐site APP‐cleaving enzyme 1 (BACE1), and γ‐secre