𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Molecular and Cellular Evidence for the Alternative Lengthening of Telomeres (ALT) Mechanism in Chicken

✍ Scribed by O’Hare, T.H.; Delany, M.E.

Book ID
119985540
Publisher
S. Karger AG
Year
2011
Tongue
French
Weight
793 KB
Volume
135
Category
Article
ISSN
1424-8581

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Evidence for alternative lengthening of
Evidence for alternative lengthening of telomeres in liposarcomas in the absence of ALT-associated PML bodies
✍ Jennie N. Jeyapalan; Aaron Mendez-Bermudez; Nadia Zaffaroni; Yuri E. Dubrova; Ni 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 French ⚖ 454 KB

## Abstract Immortalized and cancer cells maintain their telomeres by activation of a telomere maintenance mechanism (TMM). In ∼85% of cancers telomerase is activated (TA) but in some tumours, in particular sarcomas, an alternative lengthening of telomeres (ALT) pathway is used. Liposarcomas are th

Telomere maintenance in wilms tumors: Fi
Telomere maintenance in wilms tumors: First evidence for the presence of alternative lengthening of telomeres mechanism
✍ Lorenza Venturini; Maria Grazia Daidone; Rosita Motta; Paola Collini; Filippo Sp 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 486 KB

## Abstract Unlimited proliferative potential is a hallmark of cancer, and can be achieved through the activation of telomere maintenance mechanisms (TMMs). Most tumors activate telomerase, but a significant minority, mainly of mesenchymal origin, uses a recombination‐based, alternative lengthening

Divergent patterns of telomere maintenan
Divergent patterns of telomere maintenance mechanisms among human sarcomas: Sharply contrasting prevalence of the alternative lengthening of telomeres mechanism in Ewing's sarcomas and osteosarcomas
✍ Gary A. Ulaner; Andrew R. Hoffman; Jesse Otero; Hsuan-Ying Huang; Zhiquan Zhao; 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 155 KB

## Abstract Two types of telomere maintenance mechanisms (TMMs) have been described in human tumors: telomerase activation and alternative lengthening of telomeres (ALT). Although the vast majority of epithelial tumors rely on telomerase activation, many mesenchymal tumors rely on ALT for telomere