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Methylation of thep73gene in gliomas

✍ Scribed by Takao Watanabe; Huatao Huang; Mitsutoshi Nakamura; Jörg Wischhusen; Michael Weller; Paul Kleihues; Hiroko Ohgaki

Publisher
Springer-Verlag
Year
2002
Tongue
English
Weight
71 KB
Volume
104
Category
Article
ISSN
0001-6322

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BACKGROUND. p53 is the most highly mutated tumor suppressor gene in human cancers. Recently, p73, a first homologue of p53, was identified and considered to be an imprinted tumor suppressor gene. Thus, we analyzed the possible role of p73 in human prostate cancers. METHODS. We investigated the expre

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Background and Procedure. The p53 gene homologue, p73, is located on the 1p36-3 locus, which is frequently deleted in human neuroblastoma (NB). A survey of 61 NB showed that among 33% of informative cases, p73 loss of heterozygosity (LOH) occurred in 7 of 20 (35%). Results. LOH pattern of vicinal ma

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## Abstract To clarify the significance of p73 in Epstein‐Barr virus (EBV)‐associated gastric carcinoma (GC), the immunohistochemical expression and CpG‐island methylation of p73 were evaluated in cancer tissues and adjacent nonneoplastic tissues of GC with and without EBV infection. Loss of p73 ex