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Mechanism of testicular atrophy induced by Di-n-butyl phthalate in rats. Part 2. The effects on some testicular enzymes

✍ Scribed by Masamichi Fukuoka; Yu Zhou; Akira Tanaka; Isao Ikemoto; Toyohei Machida


Publisher
John Wiley and Sons
Year
1990
Tongue
English
Weight
758 KB
Volume
10
Category
Article
ISSN
0260-437X

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✦ Synopsis


Division of Biological Chcmistry. Niition.il Institute ol' Hygienic Scicnccs. Kiimiyo~~i-I-chomc. Sciaga)aLu. l'ohvo 158. Japan


πŸ“œ SIMILAR VOLUMES


Mechanism of testicular atrophy induced
✍ Masamichi Fukuoka; Tsuyoshi Tanimoto; Yu Zhou; Nana Kawasaki; Akira Tanaka; Isao πŸ“‚ Article πŸ“… 1989 πŸ› John Wiley and Sons 🌐 English βš– 740 KB

Repeated oral doses of di-n-butyl phthalate (DBP) to male rats caused a decrease in testicular fructose and glucose and a sloughing of the germ cells on the first day of treatment. On day 2, more severe sloughing was seen and was accompanied by decreases in testicular iron and zinc levels and increa

Mechanism of testicular atrophy induced
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This study reports changes in levels of ferritin, haemoglobin and transferrin in the bone marrow, liver and spleen as an attempt to determine the causes of testicular iron depletion. A single oral dose of di-n-butyl phthalate (DBP) to male rats caused a sloughing of the germ cells (at 6 h) prior to

Mechanisms of testicular atrophy induced
✍ Yu Zhou; Masamichi Fukuoka; Akira Tanaka πŸ“‚ Article πŸ“… 1990 πŸ› John Wiley and Sons 🌐 English βš– 645 KB

## Abstract A single oral dose of di‐__n__‐butyl phthalate (DBP) to male rats caused a sloughing of the germ cells at 6 h, with more severe sloughing at 24 and 48 h. DBP is metabolized to mono‐__n__‐butyl phthalate (MBP), which is transported through the blood‐tubular barrier into the seminiferous

Mechanism of testicular atrophy induced
✍ Masamichi Fukuoka; Tetsu Kobayashi; Yu Zhou; Takao Hayakawa πŸ“‚ Article πŸ“… 1993 πŸ› John Wiley and Sons 🌐 English βš– 568 KB

A single oral dose of di-n-butyl phthalate (DBP) to male rats caused a sloughing of the germ cells at 6 h both with a decrease in the activity of succinate dehydrogenase (SDH) in the Sertoli cells and in the Sertoli-germ connection and with an increase in the activity of lactate dehydrogenase (LDH)