𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Mechanism of sbcB-suppression of the recBC-deficiency in postreplication repair in UV-irradiated Escherichia coli K-12

✍ Scribed by Wang, Tzu-chien V. ;Smith, Kendric C.


Publisher
Springer
Year
1985
Tongue
English
Weight
529 KB
Volume
201
Category
Article
ISSN
0026-8925

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


The dependence of postreplication repair
✍ Rothman, Robert H. ;Clark, Alvin J. πŸ“‚ Article πŸ“… 1977 πŸ› Springer 🌐 English βš– 660 KB

Mutants carrying recF143 or recF144 show wild type levels of host cell reactivation of UV-irradiated lambdavir and wild type rates of excision gap closure in repairing UV damage to their own DNA. The same mutants showed reduced rates of postreplication repair strand joining. When uvrA- recF- or uvrB

Uvm mutants of Escherichia coli K 12 def
✍ Steinborn, Gerhard πŸ“‚ Article πŸ“… 1978 πŸ› Springer 🌐 English βš– 714 KB

Selection for defective reversion induction, after UV treatment of E. coli K 12, yielded uvm mutants. These mutants exhibited highly reduced or no UV mutability for all loci tested although they were moderately and normally mutable by X-rays and EMS, respectively. Uvm mutations confer only a slight

Uvm mutants of Escherichia coli K12 defi
✍ Steinborn, Gerhard πŸ“‚ Article πŸ“… 1979 πŸ› Springer 🌐 English βš– 459 KB

Uvm mutants of Escherichia coli K12 selected for defective UV reversion induction have previously been reported to differ considerably from the UV-reversion-less recA and lexA mutants with regard to survival or mutagenic response to UV, X-rays and alkylating agents. In the present study, the phenoty

The effect of the OmpT protease on excis
✍ Milena SedliakovΓ‘; FrantiΕ‘ek MaΕ‘ek; Viera SlezΓ‘rikovΓ‘; Miroslav PirΕ‘el πŸ“‚ Article πŸ“… 1997 πŸ› Elsevier Science 🌐 English βš– 244 KB

The extent of pyrimidine dimcr excision ( PDE ) wax inhibited in U v -irradiated F. (wli KS272 1 o.q~7" ) cells who. dkey wcre preindnced by a low UV predose preceded by a nutrition stress but not in the preinduced E.t'oli SFI(IO lompT ) mutants. The preinduction, however. markedly inhibited PDE in

Defective excision and postreplication r
✍ Rothman, Robert H. ;Clark, Alvin J. πŸ“‚ Article πŸ“… 1977 πŸ› Springer 🌐 English βš– 1007 KB

The mutation recL152 leads to a reduction of excision repair as measured by an increase in the time required to close uvrA uvrB dependent incision breaks, and by a reduction of host cell reactivation ability. Postreplication repair is also delayed when measured in a uvrB5 recL152 double mutant. Such