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Linkage analysis in von Recklinghausen neurofibromatosis (NF1) with DNA markers for chromosome 17

โœ Scribed by Seizinger, B.R.; Rouleau, G.A.; Lane, A.H.; Farmer, G.; Ozelius, L.J.; Haines, J.L.; Parry, D.M.; Korf, B.R.; Pericak-Vance, M.A.; Faryniarz, A.G.; Hobbs, W.J.; Iannazzi, J.A.; Roy, J.C.; Menon, A.; Bader, J.L.; Spence, M.A.; Chao, M.V.; Mulvihill, J.J.; Roses, A.D.; Martuza, R.L.; Breakefield, X.O.; Conneally, P.M.; Gusella, J.F.

Book ID
122356258
Publisher
Elsevier Science
Year
1987
Tongue
English
Weight
247 KB
Volume
1
Category
Article
ISSN
0888-7543

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Molecular genetic analysis of tumors in
Molecular genetic analysis of tumors in von recklinghausen neurofibromatosis: Loss of heterozygosity for chromosome 17
โœ Gary R. Skuse; Barbara A. Kosciolek; Dr. Peter T. Rowley ๐Ÿ“‚ Article ๐Ÿ“… 1989 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 474 KB

The most common inherited syndrome in man predisposing t o neoplasia is neurofibromatosis-I (von Recklinghausen disease) (NFI). We investigated the hypothesis that affected individuals carry a single inactive allele at the NFI locus in the germline and that a tumor arises from a cell in a susceptibl