𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Inhibition of herpes simplex virus reactivation by dipyridamole in a mouse model

✍ Scribed by Hay, Kathleen A.; Gaydos, Andrew; Tenser, Richard B.


Publisher
John Wiley and Sons
Year
1996
Tongue
English
Weight
726 KB
Volume
50
Category
Article
ISSN
0146-6615

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Oncolysis by viral replication and inhib
✍ John T. Mullen; James M. Donahue; Soundararajalu Chandrasekhar; Sam S. Yoon; Wen πŸ“‚ Article πŸ“… 2004 πŸ› John Wiley and Sons 🌐 English βš– 645 KB

## Abstract ## BACKGROUND In preclinical models, infection of tumors by oncolytic strains of herpes simplex virus 1 (HSV‐1) resulted in the destruction of tumor cells by viral replication and release of progeny virion that infected and destroyed adjacent tumor cells. However, complete tumor regres

Hormonal modulation of herpes simplex vi
✍ Gamil P. Sawiris; Dr. Robert J. Sydiskis; Nasir Bashirelahi πŸ“‚ Article πŸ“… 1994 πŸ› John Wiley and Sons 🌐 English βš– 497 KB

In this study, the effect of the synthetic glucocorticoid hormone dexamethasone (DXM) on HSV replication was studied in a DXM receptor-positive mouse neuroblastoma (NB) cell line. In cells treated with 10-7M DXM and then infected with HSV, there was a statistically significant 9-1 8-fold increase in

Effect of dexamethasone on herpes simple
✍ Larry L. Dreyer; Dr. Robert J. Sydiskis; Nasir Bashirelahi πŸ“‚ Article πŸ“… 1989 πŸ› John Wiley and Sons 🌐 English βš– 680 KB

Monolayers of NB41A3 (MNB) cells were exposed to pharmacologic doses of dexamethasone (DXM). After 24-h, the cells were infected with herpes simplex virus type 1 (HSV-I) at a multiplicity of infection (m.0.i.) = 0.1. After every 24-h period, extracellular and intracellular virus aliquots were collec

Inhibition by acyclovir of herpes simple
✍ Dr Louis S. Kucera; Phillip A. Furman; Gertrude B. Elion πŸ“‚ Article πŸ“… 1983 πŸ› John Wiley and Sons 🌐 English βš– 525 KB

## Abstract Rat embryo fibroblasts (REF) morphologically transformed by herpes simplex virus type 2 (HSV‐2) and tumor‐derived cells were tested for ability to grow in the presence of 9‐(2‐hydroxyethoxymethyl) guanine (acyclovir). Results indicated that the effective dose of acyclovir (ACV) required