Induction of hepatic stellate cell proliferation by LPS-stimulated peripheral blood mononuclear cells from patients with liver cirrhosis
β Scribed by Kyoko Toda; Naoki Kumagai; Kanji Tsuchimoto; Hiroyuki Inagaki; Tatsuo Suzuki; Tazuko Oishi; Kazuhiro Atsukawa; Hidetsugu Saito; Toshio Morizane; Toshifumi Hibi; Hiromasa Ishii
- Publisher
- Springer Japan
- Year
- 2000
- Tongue
- English
- Weight
- 405 KB
- Volume
- 35
- Category
- Article
- ISSN
- 0944-1174
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Since the natural history of hepatitis C virusassociated liver disease and the therapeutic responsiveness might vary according to liver and blood mononuclear cells viral levels, it may be important to quantitate viral RNA in liver, blood mononuclear cells and serum, and to compare these data with ge
We investigated the role of the interferon system in the pathogenesis of chronic liver disease. Interferon-? production by peripheral blood mononuclear cells was meaeured with an ELISA. While concanavalin A-stimdated and recombinant interleukin 2-stimulated production of interferon-y in patients wit
## Abstract The mechanisms underlying chronicity of hepatitis C virus (HCV) infection are poorly understood, but the importance of impaired viral clearance by the immune system has been suggested. The prevalence of HCV infection of peripheral blood mononuclear cells (PBMC) was in investigated in 34
We investigated the production of tumor necrosis factor a by peripheral blood mononuclear cells of patients with chronic liver disease and its association with hepatitis activity. Tumor necrosis factor a production was measured with an enzyme-linked immunosorbent assay. Tumor necrosis factor a produ
Primary biliary cirrhosis (PBC) is a disease that typically affects middle-aged women and is characterized by progressive intrahepatic cholestasis which leads ultimately to cirrhosis, portal hypertension and hepatocellular failure (1). The underlying histopathological lesion in PBC is a chronic, non