๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Gait analysis detects early changes in transgenic SOD1(G93A) mice

โœ Scribed by Christine M. Wooley; Roger B. Sher; Ajit Kale; Wayne N. Frankel; Gregory A. Cox; Kevin L. Seburn


Publisher
John Wiley and Sons
Year
2005
Tongue
English
Weight
170 KB
Volume
32
Category
Article
ISSN
0148-639X

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Peripheral axotomy slows motoneuron dege
โœ Kong, Jiming; Xu, Zuoshang ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 471 KB

Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disorder that involves motoneuron degeneration, paralysis, and death. Mutations in Cu, Zn superoxide dismutase (SOD1) are one cause of this disease. It has been a puzzle as to why mutations in SOD1, an enzyme expressed in many neuronal

Impaired SDF1/CXCR4 signaling in glial p
โœ Yongquan Luo; Haipeng Xue; Andrea C. Pardo; Mark P. Mattson; Mahendra S. Rao; Ni ๐Ÿ“‚ Article ๐Ÿ“… 2007 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 446 KB

## Abstract Mutations in the superoxide dismutase 1 (SOD1) gene are associated with familial amyotrophic lateral sclerosis (ALS), and the SOD1^G93A^ transgenic mouse has been widely used as one animal model for studies of this neurodegenerative disorder. Recently, several reports have shown that ab

Glutamate AMPA receptors change in motor
โœ Massimo Tortarolo; Giuliano Grignaschi; Novella Calvaresi; Eleonora Zennaro; Gab ๐Ÿ“‚ Article ๐Ÿ“… 2006 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 550 KB

Amyotrophic lateral sclerosis (ALS) is a fatal neurological disorder involving the selective degeneration of motor neurons. In a small proportion of patients, ALS is caused by mutations in copper/zinc superoxide dismutase (SOD1), and mice overexpressing SOD1 G93A mutant develop a syndrome that close