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Fetal and neonatal von Willebrand factor (vWF) is unusually large and similar to the vWF in patients with thrombotic thrombocytopenic purpura

✍ Scribed by Mark J. Weinstein; Rita Blanchard; Joel L. Moake; Evan Vosburgh And; Kenneth MOISE

Book ID
114711848
Publisher
John Wiley and Sons
Year
1989
Tongue
English
Weight
606 KB
Volume
72
Category
Article
ISSN
0007-1048

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## Abstract Decreased von Willebrand factor (VWF)‐cleaving protease activity (<5%) has been implicated in patients with congenital thrombotic thrombocytopenic purpura–hemolytic uremic syndrome (Upshaw‐Schulman syndrome) and associated with mutations within the __ADAMTS13__ gene. In this report, we