𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Dual role for calcium in agrin signaling and acetylcholine receptor clustering

✍ Scribed by Borges, Lucia S. ;Lee, Young ;Ferns, Michael

Publisher
John Wiley and Sons
Year
2001
Tongue
English
Weight
250 KB
Volume
50
Category
Article
ISSN
0022-3034

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Overexpression of nPKC ΞΈ is inhibitory f
Overexpression of nPKC ΞΈ is inhibitory for agrin-induced nicotinic acetylcholine receptor clustering in C2C12 myotubes
✍ Kathryn Miles; Michael Wagner πŸ“‚ Article πŸ“… 2002 πŸ› John Wiley and Sons 🌐 English βš– 176 KB

## Abstract Protein kinase C (PKC) activity has been implicated in nicotinic acetylcholine receptor (nAChR) cluster disruption but the specific PKC isoforms involved have not been identified. We first tested whether phorbol esters, which activate PKCs, regulate agrin‐induced nAChR clustering in C~2

Role for calcium from the sarcoplasmic r
Role for calcium from the sarcoplasmic reticulum in coupling muscle activity to nicotinic acetylcholine receptor gene expression in rat
✍ Adams, Larry ;Goldman, Daniel πŸ“‚ Article πŸ“… 1998 πŸ› John Wiley and Sons 🌐 English βš– 189 KB πŸ‘ 3 views

subunit RNA levels as a result of decreasing nAChR ABSTRACT: Neurally evoked muscle electrical subunit promoter activity. Finally, we show that this activity suppresses nicotinic acetylcholine receptor decreased promoter activity is mediated through the (nAChR) gene expression in extrajunctional dom

Nucleotide-mediated calcium signaling in
Nucleotide-mediated calcium signaling in rat cortical astrocytes: Role of P2X and P2Y receptors
✍ Marta Fumagalli; Roberta Brambilla; Nadia D'Ambrosi; Cinzia VolontΓ©; Michela Mat πŸ“‚ Article πŸ“… 2003 πŸ› John Wiley and Sons 🌐 English βš– 358 KB πŸ‘ 1 views

## Abstract ATP is the dominant messenger for astrocyte‐to‐astrocyte calcium‐mediated communication. Definition of the exact ATP/P2 receptors in astrocytes and of their coupling to intracellular calcium ([Ca^2+^]~i~) has important implications for brain physiology and pathology. We show that, with