𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Cytogenetics in acute leukemia

✍ Scribed by Krzysztof Mrózek; Nyla A Heerema; Clara D Bloomfield


Publisher
Elsevier Science
Year
2004
Tongue
English
Weight
488 KB
Volume
18
Category
Article
ISSN
0268-960X

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Quantitative acute leukemia cytogenetics
✍ Dr. Felix Mitelman; Sverre Heim 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 811 KB

Using literature data on cytogenetic abnormalities in 3,6 I 2 cases of acute myeloid leukemia (AML) and I ,55 I cases of acute lymphocytic leukemia (ALL), we have attempted to quantify the information value of finding the typical ALL-and AMLassociated chromosome aberrations. Sensitivity, specificity

Unusual cytogenetics in a case of acute
✍ Morse, Helvise G. ;Hays, Taru ;Odom, Lorrie F. 📂 Article 📅 1979 🏛 John Wiley and Sons 🌐 English ⚖ 268 KB

The cytogenetics in the study of a patient with acute lymphoblastic leukemia are presented. Initially, a large proportion of both unstimulated and phytohemagglutinin (PHA)-stimulated blood mitoses showed an abnormal karyotype with a 7;12 translocation and a trisomy 19. At the time of relapse, a PHA-

Acute promyelocytic leukemia: Cytogeneti
✍ Jan Fraser; P. E. Hollings; P. H. Fitzgerald; A. Day; Vivienne Clark; D. C. Heat 📂 Article 📅 1981 🏛 John Wiley and Sons 🌐 French ⚖ 629 KB

## Abstract Six patients were diagnosed as having acute promyelocytic leukemia (APL) according to FAB criteria. One patient conformed to the M3 variant. Informative cytogenetic results (G‐banding) on five of the patients showed that three of them, including the M3 variant, had the 15;17 translocati

Cytogenetic study of 130 childhood acute
✍ Leverger, Guy ;Bernheim, Alain ;Daniel, Marie-Thérèse ;Flandrin, Georges ;Schais 📂 Article 📅 1988 🏛 John Wiley and Sons 🌐 English ⚖ 511 KB

Cytogenetic studies performed on 130 consecutive childhood acute nonlymphocytic leukemias (ANLL) investigated in the same center between 1977 and 1986 are reported. The incidence of clonal chromosomal abnormalities was 68.5% with uneven distribution among the groups of the FAB nomenclature. The high