✦ LIBER ✦

Behavioral Characterization of Quinolinate-Induced Lesions of the Medial Striatum: Relevance for Huntington's Disease

✍ Scribed by J.C.S. Furtado; Michael F. Mazurek

Book ID: 115606314
Publisher: Elsevier Science
Year: 1996
Tongue: English
Weight: 187 KB
Volume: 138
Category: Article
ISSN: 0014-4886
DOI: 10.1006/exnr.1996.0054

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Neurogenesis in the striatum of the quin

Neurogenesis in the striatum of the quinolinic acid lesion model of Huntington's disease

✍ A.S Tattersfield; R.J Croon; Y.W Liu; A.P Kells; R.L.M Faull; B Connor 📂 Article 📅 2004 🏛 Elsevier Science 🌐 English ⚖ 755 KB

Electrolytic lesion of globus pallidus a

Electrolytic lesion of globus pallidus ameliorates the behavioral effects of quinolinic acid lesion of the striatum: A potential novel treatment in a rat model of Huntington's disease

✍ L. Ayalon; D. Joel; R. Tarrasch; J. Feldon; I. Weiner 📂 Article 📅 1997 🏛 Elsevier Science 🌐 English ⚖ 18 KB

Impaired learning and memory after kaini

Impaired learning and memory after kainic acid lesions of the striatum: a behavioral model of Huntington's disease

✍ Paul R. Sanberg; John Lehmann; Hans C. Fibiger 📂 Article 📅 1978 🏛 Elsevier Science 🌐 English ⚖ 384 KB

Electrolytic lesion of globus pallidus a

Electrolytic lesion of globus pallidus ameliorates the behavioral and neurodegenerative effects of quinolinic acid lesion of the striatum: a potential novel treatment in a rat model of Huntington's disease

✍ D Joel; L Ayalon; R Tarrasch; L Veenman; J Feldon; I Weiner 📂 Article 📅 1998 🏛 Elsevier Science 🌐 English ⚖ 1000 KB

The IGF-I Amino-Terminal Tripeptide Glyc

The IGF-I Amino-Terminal Tripeptide Glycine-Proline-Glutamate (GPE) Is Neuroprotective to Striatum in the Quinolinic Acid Lesion Animal Model of Huntington's Disease

✍ Tajrena Alexi; Paul E. Hughes; W.M.C. van Roon-Mom; Richard L.M. Faull; Chris E. 📂 Article 📅 1999 🏛 Elsevier Science 🌐 English ⚖ 662 KB

Deficits induced by quinolinic acid lesi

Deficits induced by quinolinic acid lesion to the striatum in a position discrimination and reversal task are ameliorated by permanent and temporary lesion to the globus pallidus: A potential novel treatment in a rat model of Huntington's disease

✍ Daphna Joel; Liat Ayalon; Ricardo Tarrasch; Ina Weiner 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 241 KB 👁 1 views

## Abstract Symptoms in the early stages of Huntington's disease (HD) are assumed to reflect basal ganglia circuit dysfunction secondary to degeneration of striatal projections to the external segment of the globus pallidus (GPe). The hypothesis that GPe lesion would ameliorate HD symptoms by “norm