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Base excision repair genesXRCC1andAPEX1and the risk for prostate cancer

✍ Scribed by H. Kuasne; I. S. Rodrigues; R. Losi-Guembarovski; M. B. Reis; P. E. Fuganti; E. P. Gregório; F. Libos Junior; H. M. Matsuda; M. A. F. Rodrigues; M. O. Kishima; I. M. S. Cólus

Publisher: Springer
Year: 2010
Tongue: English
Weight: 215 KB
Volume: 38
Category: Article
ISSN: 0301-4851
DOI: 10.1007/s11033-010-0267-z

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## Abstract ## BACKGROUND. Tobacco smoke contains numerous carcinogens that cause DNA damage, including oxidative lesions that are removed effectively by the base‐excision repair (BER) pathway, in which adenosine diphosphate ribosyl transferase (ADPRT), x‐ray repair cross‐complementing 1 (XRCC1),