𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Antibiotic resistant mutants of Escherichia coli K12 show increases in heterologous gene expression

✍ Scribed by Adnan Ahmetagic; John M. Pemberton


Book ID
113855473
Publisher
Elsevier Science
Year
2011
Tongue
English
Weight
644 KB
Volume
65
Category
Article
ISSN
0147-619X

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


cysB and cysE mutants of Escherichia col
✍ Rakonjac, Jasna ;Milic, Mirjana ;Savic, Dragutin J. πŸ“‚ Article πŸ“… 1991 πŸ› Springer 🌐 English βš– 506 KB

Mutations in the cysB and cysE genes of Escherichia coli K12 cause an increase in resistance to the gyrase inhibitor novobiocin but not to coumermycin, acriflavine and rifampicin. This unusual relationship was also observed among spontaneous novobiocin resistant (Novr) mutants: 10% of Novr mutants i

Expression of antibiotic resistance gene
✍ Kreft, JΓΌrgen ;Burger, Klaus J. ;Goebel, Werner πŸ“‚ Article πŸ“… 1983 πŸ› Springer 🌐 English βš– 689 KB

Bifunctional recombinant plasmids were constructed, comprised of the E. coli vectors pBR322, pBR325 and pACYC184 and different plasmids from Gram-positive bacteria, e.g. pBSU161-1 of B. subtilis and pUB110 and pC221 of S. aureus. The beta-lactamase (bla) gene and the chloramphenicol acetyltransferas

Viability of Escherichia coli K-12 DNA a
✍ Peterson, Kenneth R. ;Wertman, Kenneth F. ;Mount, David W. ;Marinus, M. G. πŸ“‚ Article πŸ“… 1985 πŸ› Springer 🌐 English βš– 774 KB

We have examined the level of expression of the SOS regulon in cells lacking DNA adenine methylase activity (darn-). Mud (Ap, lac) fusions to several SOS operons (recA, lexA, uvrA, uvrB, uvrD, sulA, dinD and dinF) were found to express higher levels of fl-galactosidase in darn-strains than in isogen

Functional expression of Aquaspirillum m
✍ Waleh, Nahid S. πŸ“‚ Article πŸ“… 1988 πŸ› Springer 🌐 English βš– 544 KB

Gene libraries from the magnetotactic bacterium, Aquaspirillum magnetotacticum were constructed in Escherichia coli with cosmids pLAFR3 and c2RB as vectors. Recombinant cosmids able to complement the thr-1, leuB, and proA mutations of the host were identified. The Pro+ recombinant cosmid restored wi