✦ LIBER ✦

β-Amyloid stimulates glial cells in vitro to produce growth factors that accumulate in senile plaques in Alzheimer's disease

✍ Scribed by Dalia M. Araujo; Carl W. Cotman

Book ID: 115802673
Publisher: Elsevier Science
Year: 1992
Tongue: English
Weight: 475 KB
Volume: 569
Category: Article
ISSN: 0006-8993
DOI: 10.1016/0006-8993(92)90380-r

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Secreted amyloid β–protein similar to th

Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

✍ Scheuner, D.; Eckman, C.; Jensen, M.; Song, X.; Citron, M.; Suzuki, N.; Bird, T. 📂 Article 📅 1996 🏛 Nature Publishing Group 🌐 English ⚖ 764 KB

Secreted amyloid β–protein similar to th

Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

✍ Scheuner, D.; Eckman, C.; Jensen, M.; Song, X.; Citron, M.; Suzuki, N.; Bird, T. 📂 Article 📅 1996 🏛 Nature Publishing Group 🌐 English ⚖ 764 KB

To determine whether the presenilin 1 (PS1), presenilin 2 (PS2) and amyloid beta-protein precursor (APP) mutations linked to familial Alzheimer's disease (FAD) increase the extracellular concentration of amyloid beta-protein (A beta) ending at A beta 42(43) in vivo, we performed a blinded comparison

Cytokines Associated with Amyloid Plaque

Cytokines Associated with Amyloid Plaques in Alzheimer's Disease Brain Stimulate Human Glial and Neuronal Cell Cultures to Secrete Early Complement Proteins, But Not C1-Inhibitor

✍ Robert Veerhuis; Ingrid Janssen; Corline J.A. De Groot; Freek L. Van Muiswinkel; 📂 Article 📅 1999 🏛 Elsevier Science 🌐 English ⚖ 227 KB

Processing progress in Alzheimer's disea

Processing progress in Alzheimer's disease : Inhibition of amyloid β-protein production in neural cells by the protease inhibitor AEBSF Citron, M. et al. (1996) Neuron 17, 171–179 Secreted amyloid β-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the preselinin 1 nad 2 and APP mutations linked to familial Alzheimer's disease Scheuner, D. et al. (1996) Nat. Med. 2, 864–870

✍ B.Joy Snider 📂 Article 📅 1996 🏛 Elsevier Science 🌐 English ⚖ 328 KB