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Processing progress in Alzheimer's disease : Inhibition of amyloid β-protein production in neural cells by the protease inhibitor AEBSF Citron, M. et al. (1996) Neuron 17, 171–179 Secreted amyloid β-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the preselinin 1 nad 2 and APP mutations linked to familial Alzheimer's disease Scheuner, D. et al. (1996) Nat. Med. 2, 864–870

✍ Scribed by B.Joy Snider

Book ID
113410952
Publisher
Elsevier Science
Year
1996
Tongue
English
Weight
328 KB
Volume
2
Category
Article
ISSN
1357-4310

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Secreted amyloid β–protein similar to th
Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease
✍ Scheuner, D.; Eckman, C.; Jensen, M.; Song, X.; Citron, M.; Suzuki, N.; Bird, T. 📂 Article 📅 1996 🏛 Nature Publishing Group 🌐 English ⚖ 764 KB

To determine whether the presenilin 1 (PS1), presenilin 2 (PS2) and amyloid beta-protein precursor (APP) mutations linked to familial Alzheimer's disease (FAD) increase the extracellular concentration of amyloid beta-protein (A beta) ending at A beta 42(43) in vivo, we performed a blinded comparison