Virus multiplication and induction of apoptosis by Sendai virus: role of the C proteins
✍ Scribed by A.Hajime Koyama; Hiroshi Irie; Atsushi Kato; Yoshiyuki Nagai; Akio Adachi
- Book ID
- 108489021
- Publisher
- Elsevier Science
- Year
- 2003
- Tongue
- English
- Weight
- 570 KB
- Volume
- 5
- Category
- Article
- ISSN
- 1286-4579
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Interferon (IFN) was detected upon co‐culture of cloned Sendai virus (SV)‐specific T lymphocyte with SV‐presenting syngeneic stimulator cells. The antiviral activity was defined as IFN‐α, β. The T cell clones, upon contact with antigen‐presenting stimulator cells, stimulated adherent ce
## Abstract Virus induction by association of non‐permissive cells transformed by Rous sarcoma virus (RSV) and permissive chicken cells in the absence of Sendai virus has been studied with two clones of BHK21 hamster fibroblasts transformed respectively by Schmidt‐Ruppin strain RSV (clone RS2) and
## Abstract Measles virus (MV) causes transient but profound immunosuppression resulting in increased susceptibility to secondary bacterial and viral infections. Due to the development of these opportunistic infections, measles remains the leading vaccine‐preventable cause of child death worldwide.
## Abstract Minimal numbers of CD8^+^ T cells are found in bronchoalveolar lavage (BAL) populations recovered from Sendai virus‐infected mice that are homozygous (−/−) for β2‐microglobulin (β2‐m) gene disruption. The prevalence of the CD8^+^ set was substantially increased in the pneumonic lungs of