Upregulation of Fas-induced apoptosis by interferon-γ accompanied by increased ICE expression in renal cell cancer cells
✍ Scribed by Y. Tomita; V. Bilim; M. Takeda; K. Takahashi
- Publisher
- Springer
- Year
- 1996
- Tongue
- English
- Weight
- 345 KB
- Volume
- 1
- Category
- Article
- ISSN
- 1360-8185
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
The effect of interferon-gamma (IFN-gamma) on the interaction between tumor cells and mesothelial cell layers was studied from the aspect of changes in mesothelial permeability. Mesothelial permeability was assessed as the percentage diffusion of radiolabeled albumin across the mesothelial cell shee
Background and Objectives: Fas (APO-1) induces apoptosis after binding Fas ligand (FasL). Evidence suggests that tumors may use this interaction to evade the host immune response. Fas/FasL expression has not been reported in esophageal cancer. We hypothesized that Fas expression would render esophag
## Abstract We examined the apoptosis of tongue carcinoma cells and the effects of anticancer drugs to identify the molecules that mediate apoptotic cascade in the malignancy. Carboplatin (CBDCA) induced apoptosis of SCC‐9 and SCC‐25, human well‐differentiated tongue squamous carcinoma cell lines.
## Abstract Nonsteroidal anti‐inflammatory drug‐activated gene‐1 (NAG‐1) has recently been shown to be induced by nonsteroidal anti‐inflammatory drugs (NSAIDs) and to have proapoptotic and antitumorigenic activities. Although sulindac sulfide induced apoptosis in sinonasal cancer cells, the relatio