Fanconi anemia (FA) is a genetic syndrome predisposing to hematopoietic failure. Little is known about the pathophysiology of FA, except that tumor necrosis factor-␣ (TNF-␣) is overexpressed in patients. FA group C (Fac) gene knockout mice have been developed in order to model the human disease, but
Tumor necrosis factor-α overproduction in Fanconi's anemia
✍ Scribed by John C. Schultz; Nasrollah T. Shahidi
- Publisher
- John Wiley and Sons
- Year
- 1993
- Tongue
- English
- Weight
- 576 KB
- Volume
- 42
- Category
- Article
- ISSN
- 0361-8609
No coin nor oath required. For personal study only.
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