✦ LIBER ✦

TRUSS, a tumor necrosis factor receptor-1-interacting protein, activates c-Jun NH2-terminal kinase and transcription factor AP-1

✍ Scribed by Surinder M. Soond; Jennifer L. Terry; David W.H. Riches

Book ID: 116466841
Publisher: Elsevier Science
Year: 2006
Tongue: English
Weight: 458 KB
Volume: 580
Category: Article
ISSN: 0014-5793
DOI: 10.1016/j.febslet.2006.06.098

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Suppression of tumor necrosis factor-act

Suppression of tumor necrosis factor-activated nuclear transcription factor-κB, activator protein-1, c-Jun N-terminal kinase, and apoptosis by β-lapachone

✍ Sunil K Manna; Yash P Gad; Asok Mukhopadhyay; Bharat B Aggarwal 📂 Article 📅 1999 🏛 Elsevier Science 🌐 English ⚖ 641 KB

Lack of requirement of STAT1 for activat

Lack of requirement of STAT1 for activation of nuclear factor-κB, c-Jun NH2-terminal protein kinase, and apoptosis by tumor necrosis factor-α

✍ Asok Mukhopadhyay; Shishir Shishodia; Xin-Yuan Fu; Bharat B. Aggarwal 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 377 KB

## Abstract Tumor necrosis factor (TNF) is one of the most potent activators of nuclear transcription factor NF‐κB, c‐Jun N‐terminal protein kinase (JNK), and apoptosis in a wide variety of cells. The biological effects of TNF are mediated through sequential interactions of various cytoplasmic prot

Lead Activates Nuclear Transcription Fac

Lead Activates Nuclear Transcription Factor -κB, Activator Protein-1, and Amino-Terminal c-Jun Kinase in Pheochromocytoma Cells

✍ Govindarajan T. Ramesh; Sunil K. Manna; Bharat B. Aggarwal; Arun L. Jadhav 📂 Article 📅 1999 🏛 Elsevier Science 🌐 English ⚖ 206 KB

Histamine differentially interacts with

Histamine differentially interacts with tumor necrosis factor-α and thrombin in endothelial tissue factor induction: the role of c-Jun NH2-terminal kinase

✍ J. STEFFEL; C. ARNET; A. AKHMEDOV; S. M. ISELI; T. F. LÜSCHER; F. C. TANNER 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 832 KB

Cellular resistance to vincristine suppr

Cellular resistance to vincristine suppresses NF-κB activation and apoptosis but enhances c-Jun-NH2-terminal protein kinase activation by tumor necrosis factor

✍ D. K. Giri; P. Pantazis; B. B. Aggarwal 📂 Article 📅 1999 🏛 Springer 🌐 English ⚖ 418 KB

A peptide fragment of ependymin neurotro

A peptide fragment of ependymin neurotrophic factor uses protein kinase C and the mitogen-activated protein kinase pathway to activate c-Jun N-terminal kinase and a functional AP-1 containing c-Jun and c-Fos proteins in mouse NB2a cells

✍ David S. Adams; Brendan Hasson; Anne Boyer-Boiteau; Adam El-Khishin; Victor E. S 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 376 KB 👁 1 views

## Abstract Ependymin (EPN) is a goldfish brain neurotrophic factor previously shown to function in a variety of cellular events related to long‐term memory formation and neuronal regeneration. CMX‐8933, an 8‐amino‐acid synthetic peptide fragment of EPN, was designed for aiding an investigation of