The discovery of mutations in the human SOD1 gene encoding Cu,Zn superoxide dismutase (Cu,Zn SOD) in patients with familial amyotrophic lateral sclerosis (ALS) has made possible the development of etiological models of the disease. Expression of mutant SOD1 genes in transgenic mice causes a progress
β¦ LIBER β¦
The use of transgenic mouse models of amyotrophic lateral sclerosis in preclinical drug studies
β Scribed by Mark E Gurney
- Book ID
- 119468754
- Publisher
- Elsevier Science
- Year
- 1997
- Tongue
- English
- Weight
- 94 KB
- Volume
- 152
- Category
- Article
- ISSN
- 0022-510X
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