✦ LIBER ✦

The transcription factors hypoxia-inducible factor 1α and Ets-1 colocalize in the hypoxic synovium of inflamed joints in adjuvant-induced arthritis

✍ Scribed by C. L. Peters; C. J. Morris; P. I. Mapp; D. R. Blake; C. E. Lewis; V. R. Winrow

Publisher: John Wiley and Sons
Year: 2004
Tongue: English
Weight: 163 KB
Volume: 50
Category: Article
ISSN: 0004-3591
DOI: 10.1002/art.11473

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Responses to the proinflammatory cytokin

Responses to the proinflammatory cytokines interleukin-1 and tumor necrosis factor α in cells derived from rheumatoid synovium and other joint tissues involve nuclear factor κB–mediated induction of the Ets transcription factor ESE-1

✍ Franck Grall; Xuesong Gu; Lujian Tan; Je-Yoel Cho; Mehmet Sait Inan; Allison R. 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 432 KB 👁 2 views

Organ preservation solutions attenuate a

Organ preservation solutions attenuate accumulation and nuclear translocation of hypoxia-inducible factor-1α in the hepatoma cell line HepG2

✍ Renate Paddenberg; Nele Howold; Christiane Hoger; Hermann Janßen; Veronika Grau; 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 412 KB

Hypoxia-inducible factor 1α is involved

Hypoxia-inducible factor 1α is involved in the prostaglandin metabolism of osteoarthritic cartilage through up-regulation of microsomal prostaglandin E synthase 1 in articular chondrocytes

✍ C. Grimmer; D. Pfander; B. Swoboda; T. Aigner; L. Mueller; F. F. Hennig; K. Gels 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 411 KB 👁 1 views

Interleukin-1β (IL-1β)-induced modulatio

Interleukin-1β (IL-1β)-induced modulation of the hypothalamic IL-1β system, tumor necrosis factor-α, and transforming growth factor-β1 mRNAs in obese (fa/fa) and lean (Fa/Fa) Zucker rats: Implications to IL-1β feedback systems and cytokine–cytokine interactions

✍ Carlos R. Plata-Salamán; Sergey E. Ilyin 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 111 KB 👁 1 views

## Interleukin -1␤ (IL-1␤) induces anorexia, fever, sleep changes, and neuroendocrine alterations when administered into the brain. Here, we investigated the regulation of the IL-1␤ system (ligand, receptors, receptor accessory protein, and receptor antagonist), tumor necrosis factor-␣ (TNF-␣), tr