๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

The role of X-linked FOXP3 in the autoimmune susceptibility of Turner Syndrome patients

โœ Scribed by Maureen A. Su; Matthew Stenerson; Weihong Liu; Amy Putnam; Felix Conte; Jeffrey A. Bluestone; Mark S. Anderson


Book ID
113540731
Publisher
Elsevier Science
Year
2009
Tongue
English
Weight
473 KB
Volume
131
Category
Article
ISSN
1090-2341

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


The immune dysregulation, polyendocrinop
โœ Ochs, Hans D.; Bennett, Craig L.; Christie, Jacinda; Ramsdell, Fred; Brunkow, Ma ๐Ÿ“‚ Article ๐Ÿ“… 2001 ๐Ÿ› Nature Publishing Group ๐ŸŒ English โš– 44 KB

The gene WAS, contained in this interval, was excluded as a candidate for IPEX (refs. 1,2). The scurfy syndrome in mice 3 shares phenotypic features with IPEX and maps to a region of conserved synteny on the mouse X chromosome. Human and mouse FOXP3 were recently identified by positional cloning and

Isodicentric X chromosome in a patient w
โœ Anjana Lal Pettigrew; Edward R. B. McCabe; David H. Ledbetter; Frederick F. B. E ๐Ÿ“‚ Article ๐Ÿ“… 1991 ๐Ÿ› Springer ๐ŸŒ English โš– 510 KB

Cytogenetic analyses have previously shown that the region Xq11.2-q21 is retained in all structurally abnormal X chromosomes. From these observations the conclusion has been drawn that this "critical region" on the proximal long arm of the X chromosome contains the locus controlling X-inactivation.