๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

The fate of lipopolysaccharide in cultured rat Kupffer cells

โœ Scribed by Bossuyt, H. ;Desmaretz, C. ;Wisse, E.


Publisher
Springer-Verlag
Year
1989
Weight
465 KB
Volume
58
Category
Article
ISSN
0042-6431

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Uptake and modification of 125I-lipopoly
โœ Eben S. Fox; Peter Thomas; Selwyn A. Broitman ๐Ÿ“‚ Article ๐Ÿ“… 1988 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 570 KB

While it is generally believed that hepatic clearance of lipopolysaccharide involves Kupffer cells, the mechanism involved has not been fully elucidated. This study assesses this phenomenon in terms of in vitro uptake and post-uptake modification experiments with an "' 1- labeled Salmonella minnesot

Suppression of lipopolysaccharide-induce
โœ Shamimunisa B. Mustafa; Brent D. Flickinger; Merle S. Olson ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 208 KB

Excessive nitric oxide (NO) generated by hepatic cells in response to lipopolysaccharide (LPS) and inflammatory substances (e.g., platelet-activating factor [PAF]) is a key contributor to the pathophysiological outcomes observed in the liver during sepsis. In rats subjected to liver-focused endotoxe

Endothelin association with the cultured
โœ Katherine Stephenson; Stephen A. K. Harvey; Shamimunisa B. Mustafa; Ann T. Eakes ๐Ÿ“‚ Article ๐Ÿ“… 1995 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 1009 KB

Circulating endothelin (ET) levels are elevated in conditions such as endotoxemia, hepatic ischemia-reperfusion injury, or orthotopic liver transplantation, and this potent peptide may contribute to hepatic athophysiology. We measured the surface binding of [' IIET-1 to rat Kupffer cells in primary

Interleukin 1 ฮฒ markedly stimulates nitr
โœ H Kitade; K Sakitani; K Inoue; Y Masu; N Kawada; Y Hiramatsu; Y Kamiyama; T Okum ๐Ÿ“‚ Article ๐Ÿ“… 1996 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 500 KB

absence of other cytokines or LPS. (HEPATOLOGY To investigate whether a single inflammatory cyto-1996;23:797-802.) kine could stimulate nitric oxide formation in the absence of other cytokines or lipopolysaccharide (LPS), NO was measured by the redox chemiluminescence Nitric oxide plays an important