In both hereditary hemochromatosis (HHC) and in the various forms of secondary hemochromatosis, there is a pathological expansion of total body iron stores, due mainly to an inappropriate increase in absorption of dietary iron (1-3). The liver is the major recipient of this excess absorbed iron, and
β¦ LIBER β¦
The effect of vitamin E on lipid peroxidation in the copper-deficient rat
β Scribed by Sharon Silverman; Meira Fields; Charles Lewis
- Publisher
- Elsevier Science
- Year
- 1990
- Tongue
- English
- Weight
- 394 KB
- Volume
- 1
- Category
- Article
- ISSN
- 0955-2863
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