𝔖 Bobbio Scriptorium
✦   LIBER   ✦

The critical role of methionine 35 in Alzheimer's amyloid β-peptide (1–42)-induced oxidative stress and neurotoxicity

✍ Scribed by D. Allan Butterfield; Debra Boyd-Kimball


Publisher
Elsevier Science
Year
2005
Tongue
English
Weight
455 KB
Volume
1703
Category
Article
ISSN
1570-9639

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Acetyl-L-carnitine-induced up-regulation
✍ Hafiz Mohmmad Abdul; Vittorio Calabrese; Menotti Calvani; D. Allan Butterfield 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 636 KB

Alzheimer's disease (AD) is a progressive neurodegenerative disorder characterized by loss of memory and cognition and by senile plaques and neurofibrillary tangles in brain. Amyloid-beta peptide, particularly the 42-amino-acid peptide (Abeta(1-42)), is a principal component of senile plaques and is

In vivo protective effects of ferulic ac
✍ Marzia Perluigi; Gururaj Joshi; Rukhsana Sultana; Vittorio Calabrese; Carlo De M 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 337 KB 👁 1 views

Alzheimer's disease (AD) is a neurodegenerative disorder characterized by the deposition of amyloid-beta peptide (Abeta), a peptide that as both oligomers and fibrils is believed to play a central role in the development and progress of AD by inducing oxidative stress in brain. Therefore, treatment

γ-glutamylcysteine ethyl ester-induced u
✍ Debra Boyd-Kimball; Rukhsana Sultana; Hafiz Mohmmad Abdul; D. Allan Butterfield 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 249 KB 👁 1 views

## Abstract Glutathione (GSH) is an important endogenous antioxidant found in millimolar concentrations in the brain. GSH levels have been shown to decrease with aging. Alzheimer's disease (AD) is a neurodegenerative disorder associated with aging and oxidative stress. Aβ(1–42) has been shown to in