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The choleretic effects of N-acetylglucosaminides, major urinary metabolites of ursodeoxycholic acid, in bile fistula rats

✍ Scribed by Tomomichi Iwaki; Norio Hirabayashi; Norio Miyazawa; Yoshishige Takeuchi; Kaoru Ishizaki; Hiroo Sakakura; Hiroshi Kasai; Minoru Maeda; Tsutomu Araki

Book ID
114016720
Publisher
Elsevier Science
Year
1998
Tongue
English
Weight
313 KB
Volume
363
Category
Article
ISSN
0014-2999

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πŸ“œ SIMILAR VOLUMES

Regulation of bile acid synthesis. I. Ef
Regulation of bile acid synthesis. I. Effects of conjugated ursodeoxycholate and cholate on bile acid synthesis in chronic bile fistula rat
✍ Douglas M. Heuman; Carmen R. Hernandez; Philip B. Hylemon; William M. Kubaska; C πŸ“‚ Article πŸ“… 1988 πŸ› John Wiley and Sons 🌐 English βš– 1007 KB

Bile acid synthesis is thought to be regulated by a negative feedback mechanism which is presumably dependent upon the flux of bile acids in the enterohepatic circulation. To characterize further the role of bile acids in regulation of bile acid synthesis, we have administered pure taurine or glycin

The major metabolites of ursodeoxycholic
The major metabolites of ursodeoxycholic acid in human urine are conjugated with N-acetylglucosamine
✍ Dr. Hanns-Ulrich Marschall; William James Griffiths; Ulrich GΓΆtze; Jie Zhang; Hu πŸ“‚ Article πŸ“… 1994 πŸ› John Wiley and Sons 🌐 English βš– 925 KB

Ursodeoxycholic acid (750 mg/day) was administered orally to ten healthy subjects over a period of 10 days; 24 hr urine samples were collected the day before and on the last day of the study. Urinary bile acids were extracted, separated into groups of conjugates and analyzed by gas chromatography-ma

Effect of ursodeoxycholic acid on the ki
Effect of ursodeoxycholic acid on the kinetics of the major hydrophobic bile acids in health and in chronic cholestatic liver disease
✍ Ulrich Beuers; Ulrich Spengler; Franz M. Zwiebel; Juergen Pauletzki; Sven Fische πŸ“‚ Article πŸ“… 1992 πŸ› John Wiley and Sons 🌐 English βš– 619 KB

Beneficial effects of ursodeoxycholic acid in chronic cholestatic liver diseases have been attributed to displacement of hydrophobic bile acids from the endogenous bile acid pool. To test this hypothesis, we determined pool sizes, fractional turnover rates, synthesis/input rates and serum levels of