𝔖 Bobbio Scriptorium
✦   LIBER   ✦

The BK virus in renal transplant recipients—review of pathogenesis, diagnosis, and treatment

✍ Scribed by Vikas R. Dharnidharka; Husam A. Abdulnour; Carlos E. Araya

Publisher
Springer
Year
2011
Tongue
English
Weight
336 KB
Volume
26
Category
Article
ISSN
0931-041X

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Systematic screening of BK virus by real
Systematic screening of BK virus by real-time PCR prevents BK virus associated nephropathy in renal transplant recipients
✍ Anna-Lena Hammarin; Björn Öqvist; John Wahlgren; Kerstin I. Falk 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 92 KB

## Abstract BK virus associated nephropathy occurs in approximately 5% of renal transplant recipients. Quantitation of BKV DNA in serum/plasma early in the course of disease has been suggested to be an important diagnostic tool for polymavirus‐associated nephropathy (PVAN). The aim of this study wa

A prospective longitudinal study of BK v
A prospective longitudinal study of BK virus infection in 120 Czech renal transplant recipients
✍ Eva Girmanova; Irena Brabcova; Stepan Bandur; Petra Hribova; Jelena Skibova; Ond 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 147 KB

## Abstract Polyomavirus BK (BKV) is a common human polyomavirus that rarely causes clinical symptoms in immunocompetent individuals. However, BK virus reactivation occurs in 20–40% of kidney transplant patients and 1–10% of cases present with BK virus‐associated nephropathy (BKVN) and reduced kidn

Distribution of BK polyomavirus genotype
Distribution of BK polyomavirus genotypes in Tunisian renal transplant recipients
✍ Hanen Boukoum; Imen Nahdi; Vincent Foulongne; Dorsaf Zallema; Sabra Aloui; Abdel 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 128 KB

BK polyomavirus (BKV) is a ubiquitous virus in humans that remains latent in the urogenital tract after a primary infection during childhood. The virus, which is reactivated frequently and excreted in urine, can cause nephropathy in renal transplant recipients. BKV sequences are classified into four

Mutations in the external loops of BK vi
Mutations in the external loops of BK virus VP1 and urine viral load in renal transplant recipients
✍ Sara Tremolada; Serena Delbue; Lorenzo Castagnoli; Sara Allegrini; Umberto Migli 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 92 KB

## Abstract Polyomavirus‐associated nephropathy (PVAN) is a major complication that occurs after renal transplantation and is induced by reactivation of the human polyomavirus BK (BKV). The structure of the viral capsid protein 1 (VP1) is characterized by the presence of external loops, BC, DE, EF,

Erratum: A prospective longitudinal stud
Erratum: A prospective longitudinal study of BK virus infection in 120 Czech renal transplant recipients
✍ Eva Girmanova; Irena Brabcova; Stepan Bandur; Petra Hribova; Jelena Skibova; Ond 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 19 KB

The following sentences from the article abstract are corrected as follows: BKVN was confirmed histologically in 3/120 patients and viremic patients were treated with dialysis for longer time periods and had higher levels of panel reactive antibodies. In conclusion, polyoma BK viremia and viruria