𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Suppression of Cell Proliferation by Inhibition of Estrone-3-Sulfate Transporter in Estrogen-Dependent Breast Cancer Cells

✍ Scribed by Takashi Nozawa; Masato Suzuki; Hikaru Yabuuchi; Masanori Irokawa; Akira Tsuji; Ikumi Tamai

Publisher
Springer US
Year
2005
Tongue
English
Weight
446 KB
Volume
22
Category
Article
ISSN
0724-8741

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Inhibition of cell proliferation in huma
Inhibition of cell proliferation in human breast tumor cells by antisense oligonucleotides against facilitative glucose transporter 5
✍ K.K. Chan; Judy Y.W. Chan; Karen K.W. Chung; Kwok-Pui Fung πŸ“‚ Article πŸ“… 2004 πŸ› John Wiley and Sons 🌐 English βš– 196 KB πŸ‘ 1 views

## Abstract In recent years, successful examples of antisense oligonucleotide (AS) therapy for genetic diseases have stimulated scientists to investigate its application on cancer diseases. AS can be used to down‐regulate the mRNA and protein expression by annealing to specific region of the target

Homeobox gene HOPX is epigenetically sil
Homeobox gene HOPX is epigenetically silenced in human uterine endometrial cancer and suppresses estrogen-stimulated proliferation of cancer cells by inhibiting serum response factor
✍ Shinichiro Yamaguchi; Kazuo Asanoma; Tomoka Takao; Kiyoko Kato; Norio Wake πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 French βš– 687 KB

## Abstract __HOPX (homeodomain only protein X__) is a newly identified homeobox gene whose loss of expression has been reported for several types of neoplasm. Although we found most human uterine endometrial cancers (HEC) defective in HOPX expression, genetic mutations in the __HOPX__ gene were un

Calcium-dependent inhibition of polo-lik
Calcium-dependent inhibition of polo-like kinase 3 activity by CIB1 in breast cancer cells
✍ Meghna U. Naik; Ngoc T. Pham; Kristin Beebe; Wei Dai; Ulhas P. Naik πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 French βš– 699 KB

## Abstract Members of the polo‐like kinases (Plk1, Plk2, Plk3 and Plk4) are involved in the regulation of various stages of the cell cycle and have been implicated in cancer progression. Unlike its other family members, the expression of Plk3 remains steady during cell cycle progression, suggestin