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Support for an association of the C939T polymorphism in the human DRD2 gene with tardive dyskinesia in schizophrenia

✍ Scribed by Geng-Han Mo; Ding-Lieh Liao; I-Ching Lai; Ying-Chieh Wang; Jen-Yeu Chen; Chih-Yuan Lin; Tzu-Ting Chen; Mao-Liang Chen; Ya-Mei Bai; Chao-Cheng Lin; Ying-Jay Liou

Book ID
119363194
Publisher
Elsevier Science
Year
2007
Tongue
English
Weight
104 KB
Volume
97
Category
Article
ISSN
0920-9964

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## Abstract ## Objective Tardive dyskinesia (TD) is a long‐term adverse effect of antipsychotic. We evaluated candidate functional polymorphism of the G protein __Ξ²__3 subunit (GNB3) gene for association with drug‐induced TD in the Korean schizophrenic patients. ## Methods We investigated whethe

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## Abstract Tardive dyskinesia (TD) occurs in approximately 20% of patients exposed to long‐term antipsychotic treatment and may be influenced by genetic predisposition, in addition to clinical risk factors. In this study, we implemented a two‐step approach to identify susceptibility genes for TD.