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Skeletal unloading induces resistance to insulin-like growth factor I on bone formation

✍ Scribed by T Sakata; B.P Halloran; H.Z Elalieh; S.J Munson; L Rudner; L Venton; D Ginzinger; C.J Rosen; D.D Bikle

Book ID: 117807850
Publisher: Elsevier Science
Year: 2003
Tongue: English
Weight: 426 KB
Volume: 32
Category: Article
ISSN: 8756-3282
DOI: 10.1016/s8756-3282(03)00088-7

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Insulin-like growth factor (IGF) I, a polypeptide synthesized by skeletal cells, is presumed to act as an autocrine regulator of bone formation. IGF I stimulates bone replication of preosteoblastic cells and enhances the differentiated function of the osteoblast. The synthesis of skeletal IGF I is r