Severe Epstein–Barr virus-associated hemophagocytic syndrome in six adult patients
✍ Scribed by Anat Scheiman Elazary; Dana G. Wolf; Gail Amir; Batia Avni; Deborah Rund; Dina Ben Yehuda; Sigal Sviri
- Book ID
- 116631213
- Publisher
- Elsevier Science
- Year
- 2007
- Tongue
- English
- Weight
- 352 KB
- Volume
- 40
- Category
- Article
- ISSN
- 1386-6532
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## Abstract Expression of different panels of latent gene transcripts is controlled by usage of three distinct Epstein‐Barr virus (EBV) nuclear antigen (EBNA) promoters (Wp, Cp, and Qp). EBV‐associated hemophagocytic syndrome, which is often a fatal disease and generally occurs after primary EBV in
Virus-associated hemophagocytic syndrome (VAHS) has been thought to be a distinct clinical entity, characterized by intermittent fever, enlarged liver and spleen, and the appearance of hemophagocytosis. Hemopoietic cells are actively ingested by monocytes/macrophages in various organs, including lym