๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Serum protein S-100b in acute liver failure: Results of the US acute liver failure study group

โœ Scribed by Javier Vaquer; Queralt Jordano; William M. Lee; Andres T. Blei


Publisher
John Wiley and Sons
Year
2003
Tongue
English
Weight
50 KB
Volume
9
Category
Article
ISSN
1527-6465

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Acetaminophen and the U.S. acute liver f
โœ William M. Lee ๐Ÿ“‚ Article ๐Ÿ“… 2004 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 77 KB ๐Ÿ‘ 2 views

Acetaminophen overdose is the leading cause for calls to Poison Control Centers (>100,000/year) and accounts for more than 56,000 emergency room visits, 2,600 hospitalizations, and an estimated 458 deaths due to acute liver failure each year. Data from the U.S. Acute Liver Failure Study Group regist

Drug-induced acute liver failure: Result
โœ Reuben, Adrian (author);Koch, David G. (author);Lee, William M. (author) ๐Ÿ“‚ Article ๐Ÿ“… 2010 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 163 KB ๐Ÿ‘ 1 views

and the Acute Liver Failure Study Group Acute liver failure (ALF) due to drug-induced liver injury (DILI), though uncommon, is a concern for both clinicians and patients. The Acute Liver Failure Study Group has prospectively collected cases of all forms of acute liver failure since 1998. We describe

Immunonephelometric quantification of gr
โœ Wians, F H ;Lin, W ;Brown, L P ;Schiodt, F V ;Lee, W M ๐Ÿ“‚ Article ๐Ÿ“… 1997 ๐Ÿ› Wiley (John Wiley & Sons) ๐ŸŒ English โš– 105 KB

Serum levels of group-specific component (Gc) protein are useful in evaluating the likelihood of survival in patients with acute liver failure (ALF) who may be candidates for liver transplant surgery. Most methods for quantifying Gc protein concentration are either isotopic, manual, technically dema

Acetaminophen-induced acute liver failur
โœ William J. Holubek; Susanne Kalman; Robert S. Hoffman ๐Ÿ“‚ Article ๐Ÿ“… 2006 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 42 KB ๐Ÿ‘ 2 views

In addition to the hypothetical liver vulnerability of homozygous CCR5-โŒฌ32 patients, it seems that CCR5 de ficiency may increase the susceptibility to other diseases. Preliminary results from our laboratory also suggest that CCR5-deficient mice disclose higher susceptibility to experimental pancrea