It is well documented that glucocorticoid (GC) can induce the transcription of the IB␣ gene in several cell lines. GC treatment then increases IB␣ protein levels and markedly reduces the amount of active NF-B. NF-B is a strong activator of HIV-1 promoter, but the mechanism by which GC influence the
Selenium-mediated inhibition of transcription factor NF-κ B and HIV-1 LTR promoter activity
✍ Scribed by Vassili Makropoulos; Thomas Brüning; K. Schulze-Osthoff
- Publisher
- Springer-Verlag
- Year
- 1996
- Tongue
- English
- Weight
- 164 KB
- Volume
- 70
- Category
- Article
- ISSN
- 0340-5761
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Background Phosphatidylinositol (PI) 3-kinase is activated by a variety of growth factor receptors and the PI 3-kinase/Akt signaling pathway is a key regulator of cell proliferation and survival. The downstream targets of PI 3-kinase/Akt signaling include direct regulators of cell cycle progress
## Abstract We report here a role for cyclic AMP‐responsive element‐binding protein‐1 (CREB‐1) in B cell antigen receptor (BCR)‐induced growth inhibition by IFN‐γ. BCR‐induced proliferation is negatively regulated by IFN‐γ. Stimulation through BCR resulted in dose‐dependent induction of CREB‐1 bind
Thrombin, a serine protease, is an important effector of many cellular processes and has been shown to upregulate the expression of several genes. The mechanisms underlying thrombin-mediated regulation of gene transcription remain poorly understood. The original aim of this work was to study the eff
## Abstract __This article has been retracted at the request of__: Editor‐in‐Chief and Author ‘Inhibition of nuclear factor κb activity by genistein is mediated __via__ Notch‐1 signaling pathway in pancreatic cancer cells’, by Wang, Z., Zhang, Y., Banerjee, S., Li, Y. and Sarkar, F. H. The above