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Role of sepiapterin reductase gene at the PARK3 locus in Parkinson's disease

✍ Scribed by Manu Sharma; Demetrius M. Maraganore; John P.A. Ioannidis; Olaf Riess; Jan O. Aasly; Grazia Annesi; Nadine Abahuni; Anna Rita Bentivoglio; Alexis Brice; Christine Van Broeckhoven; Marie-Christine Chartier-Harlin; Alain Destée; Ana Djarmati; Alexis Elbaz; Matthew Farrer; Carlo Ferrarese; J. Mark Gibson; Suzana Gispert; Nobutaka Hattori; Barbara Jasinska-Myga; Christine Klein; Suzanne Lesage; Timothy Lynch; Peter Lichtner; Jean-Charles Lambert; Anthony E. Lang; George D. Mellick; Francesa De Nigris; Grzegorz Opala; Aldo Quattrone; Chiara Riva; Ekaterina Rogaeva; Owen A. Ross; Wataru Satake; Peter A. Silburn; Jessie Theuns; Tatsushi Toda; Hiroyuki Tomiyama; Ryan J. Uitti; Karin Wirdefeldt; Zbigniew Wszolek; Thomas Gasser; Rejko Krüger

Book ID
116781728
Publisher
Elsevier Science
Year
2011
Tongue
English
Weight
376 KB
Volume
32
Category
Article
ISSN
0197-4580

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## Abstract Familial Parkinsonism (__PARK__) genes are strong candidates for conferring susceptibility to common forms of PD. However, most studies to date have provided little evidence that their common variants substantially influence disease risk. Recently, mutations were described in the gene,