𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Recurrent postinfantile syncytial giant cell hepatitis after orthotopic liver transplantation

✍ Scribed by Jan P. Lerut; Nicolas Claeys; Olga Ciccarelli; Roberto Pisa; Christine Galant; Pierre-François Laterre; Ugo Palazzo

Book ID
110915569
Publisher
Springer
Year
1998
Tongue
English
Weight
621 KB
Volume
11
Category
Article
ISSN
0934-0874

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Recurrent hepatic lymphangiomatosis afte
Recurrent hepatic lymphangiomatosis after orthotopic liver transplantation
✍ Seong H. Ra; Robert F. Bradley; Michael C. Fishbein; Ronald W. Busuttil; David S 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 453 KB

Hepatic lymphangiomatosis is a rare disease characterized by an abnormal lymphatic proliferation involving the liver alone, liver and spleen, or multiple organs. Hepatic lymphangiomatosis becomes symptomatic secondary to compression or replacement of the normal parenchyma, which can lead to liver fa

Recurrence of hepatitis C virus infectio
Recurrence of hepatitis C virus infection after orthotopic liver transplantation
✍ Paul Martin; Santiago J. Muñoz; Adrian M. Di Bisceglie; Raphael Rubin; Jeanne G. 📂 Article 📅 1991 🏛 John Wiley and Sons 🌐 English ⚖ 375 KB 👁 2 views

## Identification of the hepatitis C virus-the main cause of posttransfusion and sporadic non -A, non-B hepatitisand the development of a diagnostic serological test have allowed us to study possible recurrence of this type of hepatitis after liver transplantation. Six of 34 consecutive transplant

Early hepatic stellate cell activation p
Early hepatic stellate cell activation predicts severe hepatitis C recurrence after liver transplantation
✍ Samer Gawrieh; Bettina G. Papouchado; Lawrence J. Burgart; Shogo Kobayashi; Mich 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 159 KB

Only a subset of hepatitis C virus (HCV)-infected patients develop progressive hepatic fibrosis after liver transplantation (LT). Hepatic stellate cell (HSC) activation is a pivotal step in hepatic fibrosis and precedes clinically apparent fibrosis. We determined whether early HSC activation, measur