𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Recombinant C5a enhances interleukin 1 and tumor necrosis factor release by lipopolysaccharide-stimulated monocytes and macrophages

✍ Scribed by Jean-Marc Cavaillon; Catherine Fitting; Nicole Haeffner-Cavaillon


Publisher
John Wiley and Sons
Year
1990
Tongue
English
Weight
417 KB
Volume
20
Category
Article
ISSN
0014-2980

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Stimulation of the hyaluronic acid level
✍ Debra M. Butler; Gerard F. Vitti; Tali Leizer; John A. Hamilton 📂 Article 📅 1988 🏛 John Wiley and Sons 🌐 English ⚖ 873 KB

Monocyte/macrophage polypeptides (monokines) alter the properties of synovial cells. This interaction could explain some of the properties of the inflamed synovium in rheumatic disease. Only recently has it been possible to test the action of purified monokines on the target synovial cells. We repor

Interleukin-4 inhibits granulocyte-macro
✍ Michael C. D. Trindade; Yasuharu Nakashima; Martin Lind; Doo-Hoon Sun; Stuart B. 📂 Article 📅 1999 🏛 Elsevier Science 🌐 English ⚖ 779 KB

## Abstract The outcome of total joint arthroplasty is determined by biological events at the bone‐implant interface. Macrophages phagocytose implant or wear debris at the interface and release proinflammatory mediators such as interleukins 1 and 6, tumor necrosis factor‐alpha, and prostaglandin E~

Enhanced Production Of Interleukin-1 And
✍ Hisanori Umehara; Shunichi Kumagai; Masao Murakami; Toshihiko Suginoshita; Koich 📂 Article 📅 1990 🏛 John Wiley and Sons 🌐 English ⚖ 376 KB 👁 1 views

Cultured peripheral blood mononuclear cells from 20 patients with scleroderma were shown to produce high levels of interleukin-la, interleukin-lp, and tumor necrosis factor a. There were positive correlations between the production of these 3 molecules. Purified monocytes from these patients produce

Interleukin-18 enhances monocyte tumor n
✍ Sheng-Ming Dai; Hiroaki Matsuno; Hiroshi Nakamura; Kusuki Nishioka; Kazuo Yudoh 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 266 KB 👁 1 views

## Abstract ## Objective At sites of inflammation, T cells exert pathologic effects through direct contact with monocyte/macrophages, inducing massive up‐regulation of interleukin‐1 (IL‐1) and tumor necrosis factor α (TNFα). We examined the regulatory effects of IL‐18 on monocyte activation by dir