๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Promoter hypermethylation and homozygous deletion of P16INK4A in biliary tract cancer

โœ Scribed by Karel Caca; Juergen Faisthammel; Andrea Tannapfel; Joachim Moessner; Frieder Berr


Book ID
119425374
Publisher
Elsevier Science
Year
2000
Tongue
English
Weight
167 KB
Volume
118
Category
Article
ISSN
0016-5085

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Homozygous deletions of p16INK4 occur fr
โœ Yahya Tamimi; Pierre Paul Bringuier; Frank Smit; Adrie van Bokhoven; Ahmed Abbas ๐Ÿ“‚ Article ๐Ÿ“… 1996 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 644 KB

We have studied p16(INK4) mutation (by PCR-SSCP) and deletion (by Southern blotting and/or multiplex PCR) in a series of 47 bilharziasis-associated tumors from Egypt and compared the results with those obtained on a series of 17 established bladder cell lines and non-bilharziasis-associated bladder

Homozygous deletion of p16INK4a and toba
โœ Kim S. Kraunz; Heather H. Nelson; Miriam Lemos; John J. Godleski; John K. Wienck ๐Ÿ“‚ Article ๐Ÿ“… 2005 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 149 KB

Inactivation of p16(INK4a) in the Rb pathway is among the most common somatic alterations observed in nonsmall cell lung cancers (NSCLCs). While epigenetic inactivation of the p16(INK4a) gene promoter has been shown to be associated with increased tobacco carcinogen exposure, little investigation of

Frequent p16INK4a promoter hypermethylat
โœ Ming-Fang Wu; Ya-Wen Cheng; Ji-Ching Lai; Min-Chih Hsu; Jung-Ta Chen; Wen-Shan L ๐Ÿ“‚ Article ๐Ÿ“… 2004 ๐Ÿ› John Wiley and Sons ๐ŸŒ French โš– 435 KB

## Abstract Inactivation of p16INK4a gene through promoter hypermethylation has been frequently observed in non small cell lung cancer; however, various studies have shown a controversial correlation between p16INK4a hypermethylation and cigarette smoking. Our recent report showed that human papill