I read with great interest the article by Das et al. 1 Although presence of nonalcoholic fatty liver (NAFL) in nonobese individuals is a fairly common observation in India, this is the first such scientific documentation for the same. However, I would like to make a few points in this regard. First
Progressive neurologic disability in methylmalonic acidemia despite transplantation of the liver
β Scribed by William L. Nyhan; Jay J. Gargus; Karen Boyle; Rick Selby; Richard Koch
- Publisher
- Springer
- Year
- 2002
- Tongue
- English
- Weight
- 60 KB
- Volume
- 161
- Category
- Article
- ISSN
- 0340-6997
No coin nor oath required. For personal study only.
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Hepatitis C recurs universally after liver transplantation (LT). Whether its progression differs after live donor liver transplantation (LDLT) and deceased donor liver transplantation (DDLT) is still debated. We retrospectively analyzed 201 consecutive LTs performed at our institution for hepatitis
Recurrent hepatitis C after liver transplantation (LT) is a major problem, since up to 30% of patients develop cirrhosis only 5 years after LT in the absence of antiviral therapy. The aim of this study was to examine the rate of progression of fibrosis and its associated risk factors in patients sub
Antiviral therapy after liver transplantation (LT) using interferon (IFN) and ribavirin (RBV) can achieve a sustained virological response (SVR) rate ranging from 20% to 45%. The aims of our study were to assess efficacy and tolerability of therapy, effect on fibrosis progression and the importance