𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Polyendocrine autoimmunity

✍ Scribed by Jennifer M. Barker

Book ID
107536169
Publisher
Current Science Inc.
Year
2005
Tongue
English
Weight
313 KB
Volume
5
Category
Article
ISSN
1534-4827

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Slow metabolic deterioration towards dia
Slow metabolic deterioration towards diabetes in islet cell antibody positive patients with autoimmune polyendocrine disease
✍ R. Wagner; S. Genovese; E. Bosi; F. Becker; P. J. Bingley; E. Bonifacio; K. A. M πŸ“‚ Article πŸ“… 1994 πŸ› Springer 🌐 English βš– 730 KB

We studied metabolic progression to IDDM in a cohort of adults who are ICA-positive and have associated autoimmune endocrine disease or circulating organ-specific autoantibodies (the Polyendocrine Study). Of the 186 individuals recruited 27 developed overt diabetes after a median follow-up of 4.5 ye

Increased IL-17A secretion in response t
Increased IL-17A secretion in response to Candida albicans in autoimmune polyendocrine syndrome type 1 and its animal model
✍ Kerstin M. Ahlgren; Silvia Moretti; Brita ArdesjΓΆ Lundgren; Iulia Karlsson; Erik πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 375 KB

## Abstract Autoimmune polyendocrine syndrome type 1 (APS‐1) is a multiorgan autoimmune disease caused by mutations in the autoimmune regulator (AIRE) gene. Chronic mucocutaneous candidiasis, hypoparathyroidism and adrenal failure are hallmarks of the disease. The critical mechanisms causing chroni