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Platelet-activating factor augments lipopolysaccharide-induced nitric oxide formation by rat Kupffer cells

✍ Scribed by SB Mustafa; KM Howard; MS Olson

Book ID
118686263
Publisher
John Wiley and Sons
Year
1996
Tongue
English
Weight
407 KB
Volume
23
Category
Article
ISSN
0270-9139

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πŸ“œ SIMILAR VOLUMES

Platelet-activating factor augments lipo
Platelet-activating factor augments lipopolysaccharide-induced nitric oxide formation by rat Kupffer cells
✍ S B Mustafa; K M Howard; M S Olson πŸ“‚ Article πŸ“… 1996 πŸ› John Wiley and Sons 🌐 English βš– 404 KB

Acute endotoxic shock is accompanied by an increase sion, vascular injury, disseminated intravascular coagin the production of nitric oxide (NO) by several differulation, and damage to numerous organs. 1 These bioent hepatic cell types. Platelet-activating factor (PAF) is logical responses are thoug

Suppression of lipopolysaccharide-induce
Suppression of lipopolysaccharide-induced nitric oxide synthase expression by platelet-activating factor receptor antagonists in the rat liver and cultured rat kupffer cells
✍ Shamimunisa B. Mustafa; Brent D. Flickinger; Merle S. Olson πŸ“‚ Article πŸ“… 1999 πŸ› John Wiley and Sons 🌐 English βš– 208 KB

Excessive nitric oxide (NO) generated by hepatic cells in response to lipopolysaccharide (LPS) and inflammatory substances (e.g., platelet-activating factor [PAF]) is a key contributor to the pathophysiological outcomes observed in the liver during sepsis. In rats subjected to liver-focused endotoxe