𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Photoaffinity labeling of the DDT1MF-2 cell α1-adrenergic receptor

✍ Scribed by Lawrence E. Cornett; James S. Norris

Publisher
Springer
Year
1985
Tongue
English
Weight
554 KB
Volume
67
Category
Article
ISSN
0300-8177

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Glucocorticoid induction of β-adrenergic
Glucocorticoid induction of β-adrenergic receptors in the DDT1MF-2 smooth muscle cell line involves synthesis of new receptor
✍ James S. Norris; Pamela Brown; Jeffrey Cohen; Lawrence E. Cornett; Peter O. Kohl 📂 Article 📅 1987 🏛 Springer 🌐 English ⚖ 529 KB

We have shown that glucocorticoids induce the appearance of beta 2-adrenergic receptors in membranes of the ductus deferens smooth muscle cell line (DDT1 MF-2). A concomitant increase in isoproterenol stimulated adenylate cyclase activity in the absence of exogenously applied GTP was observed as was

A mechanism for testosterone modulation
A mechanism for testosterone modulation of alpha-1 adrenergic receptor expression in the DDT1MF-2 smooth muscle myocyte
✍ Mark Phillippe; Trevania Saunders; Shrikar Bangalore 📂 Article 📅 1991 🏛 Springer 🌐 English ⚖ 927 KB

Previous reports have confirmed that steroid hormones modulate the expression of adrenergic receptors on the surface of smooth muscle myocytes. The present study was undertaken to evaluate the mechanism by which testosterone modulates alpha-1 adrenergic receptor expression in the DDT1 MF-2 transform

Arachidonic acid mediates non-capacitati
Arachidonic acid mediates non-capacitative calcium entry evoked by CB1-cannabinoid receptor activation in DDT1 MF-2 smooth muscle cells
✍ Dirk G. Demuth; Effimia Gkoumassi; Melloney J. Dröge; Bart G.J. Dekkers; Henk J. 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 219 KB

## Abstract Cannabinoid CB~1~‐receptor stimulation in DDT~1~ MF‐2 smooth muscle cells induces a rise in [Ca^2+^]~i~, which is dependent on extracellular Ca^2+^ and modulated by thapsigargin‐sensitive stores, suggesting capacitative Ca^2+^ entry (CCE), and by MAP kinase. Non‐capacitative Ca^2+^ entr