This article addresses the controversial issue of whether non-smokers' exposure to environmental tobacco smoke (ETS) increases their risk of developing coronary heart disease (CHD). Glantz and Parmley purport to provide toxicological and epidemiologic evidence in support of their contention that non
Phenotypic and physiologic variability in nasal epithelium cultured from smokers and non-smokers exposed to secondhand tobacco smoke
โ Scribed by Johnny L. Carson; Tsui-Shan Lu; Luisa Brighton; Milan Hazucha; Ilona Jaspers; Haibo Zhou
- Book ID
- 107427266
- Publisher
- Society for In Vitro Biology
- Year
- 2010
- Tongue
- English
- Weight
- 188 KB
- Volume
- 46
- Category
- Article
- ISSN
- 1475-2689
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๐ SIMILAR VOLUMES
This article addresses the claim by Gross in this issue that estimates of coronary heart disease deaths due to environmental tobacco smoke are indefensible. Gross's interpretation of his data is questioned on statistical inference grounds. The three new studies which he introduces are discussed and
I appreciate the opportunity to respond to Professor Gross, since it seems, from his reply, that several things may not have been clear in my original rejoinder. However, before beginning, it is worth mentioning that another prospective study of CHD and ETS exposure recently appeared (Kawachi et al.
This response is in two parts. First, the critique of Ellison and Morrison (1998) is addressed. This is followed, then, by addressing Brown's (1998) criticisms. While I am appreciative of the eorts put forth in these rejoinders to Gross (1998), I cannot, in any good